Deactivation Mechanisms of Rod Phototransduction

NIH RePORTER · NIH · R01 · $160,326 · view on reporter.nih.gov ↗

Abstract

Project Summary Retinal rod and cone photoreceptor cells mediate the essential first steps in vision by signaling light intensity and its modulation. Although the initial biochemical steps that transduce light into electrical signals in the outer segments of photoreceptors are well understood, much less is known about how outer segment currents are transformed by voltage-sensitive conductances and influenced by adaptation and circadian regulation in vivo. The work proposed in this application will investigate photoreceptor physiology the living eye and the mechanisms by which photoreceptor physiology is modulated to influence visual sensitivity and kinetics, and to mitigate photoreceptor stress. Using primarily a combination of ex vivo and in vivo electrophysiological approaches, three aims will be investigated: 1. Determine the role of Kv2.1 channels in rods. 2. Define the mechanisms that constrain photoreceptor sensitivity and kinetics in vivo. 3. Determine how photoreceptor properties are modulated by the circadian clock. ! ! !

Key facts

NIH application ID
10039505
Project number
3R01EY014047-18S1
Recipient
UNIVERSITY OF CALIFORNIA AT DAVIS
Principal Investigator
MARIE E BURNS
Activity code
R01
Funding institute
NIH
Fiscal year
2020
Award amount
$160,326
Award type
3
Project period
2002-05-01 → 2022-04-30