Neuroinflammation, Neuronal IL-1R1, and Behavior

NIH RePORTER · NIH · R01 · $342,167 · view on reporter.nih.gov ↗

Abstract

Project Summary Neuroinflammation is a significant contributor to most CNS disorders including neurodegenerative diseases and psychological disorders. A hallmark of neuroinflammation is the increased expression of the proinflammatory cytokine interleukin-1 (IL-1) in the brain. How IL-1 causes neuro- and psycho- pathologies is poorly understood. We have recently discovered that the receptor for IL-1, IL-1R1, is expressed in specific sets of neurons in the brain. These neuronal IL-1R1s, nIL-1R1, modulate neuronal activity via non-canonical signaling pathways to alter neuronal function and circuit connectivity. This application is designed to: 1) map nIL-1R1 distribution in the brain, 2)demonstrate the involvement of nIL-1R1 in neuroinflammation induced behavioral deficits and neuropathology, and 3) elucidate the mechanisms of nIL-1R1 mediated neuromodulation.

Key facts

NIH application ID
10131281
Project number
5R01NS116914-02
Recipient
FLORIDA ATLANTIC UNIVERSITY
Principal Investigator
Ning Quan
Activity code
R01
Funding institute
NIH
Fiscal year
2021
Award amount
$342,167
Award type
5
Project period
2020-04-01 → 2025-03-31