Defining the cause and consequence of aging-specific platelet specification from hematopoietic stem cells

NIH RePORTER · NIH · R01 · $550,841 · view on reporter.nih.gov ↗

Abstract

PROJECT SUMMARY/ABSTRACT Our goal is to understand platelet (Plt) production and function upon aging. Plts play essential roles in hemostasis, the process of preventing bleeding, and also participate in immune regulation. Aging is associated with a dramatic increase in platelet-related disorders, including alterations in Plt numbers (thrombocytosis or thrombocytopenia) and in Plt activity. Plts have a very short half-life and are continually produced by hematopoietic stem cells (HSCs). We have discovered that the differentiation pathways of Plt production are different in young and old mice. Here, we propose to investigate the molecular and cellular mechanisms behind the aging-specific differentiation path, and the consequences for Plt function and aging physiology. We hypothesize that the aging-specific Plt pathway contributes to the dramatically increased risk for Plt-related disorders in the elderly. Our discovery of a new, age-specific differentiation pathway provides a unique opportunity for novel discoveries towards mitigating Plt-related disorders, including thrombosis and cardiovascular disease, in the elderly.

Key facts

NIH application ID
10144356
Project number
5R01AG062879-03
Recipient
UNIVERSITY OF CALIFORNIA SANTA CRUZ
Principal Investigator
CAMILLA FORSBERG
Activity code
R01
Funding institute
NIH
Fiscal year
2021
Award amount
$550,841
Award type
5
Project period
2019-09-15 → 2024-04-30