Dietary Lipids and Silica-Triggered Autoimmunity.

NIH RePORTER · NIH · R01 · $416,246 · view on reporter.nih.gov ↗

Abstract

While heredity is a primary predisposing factor for autoimmunity, cumulative exposures to environmental factors such as toxic stressors and diet greatly impact latency and severity autoimmune diseases such as lupus. Our goal is to understand how lupus triggering by an ubiquitous environmental toxicant can be prevented by dietary modulation of cellular lipids. This goal is predicated on preliminary studies revealing that 1)airway exposure to crystalline silica, a recognized autoimmune disease risk factor, triggers early onset of systemic autoimmunity and glomerulonephritis in the lupus-prone female NZBWF1 mouse and 2)supplementing their diets with the omega-3 fatty acid docosahexaenoic acid (DHA), a well-known dietary supplement extracted from cold-water fish, dose-dependently blocks this triggering. Here, we will employ in vitro, ex vivo, and in vivo approaches to test the hypothesis that DHA consumption prevents silica- triggered autoimmunity by suppressing inflammasome activation, cell death and self-antigen release in alveolar macrophages of NZBWF1 mice. Revealing DHA mechanisms against silica- triggered lupus will bring novel insights into how respiratory toxicants initiate lupus and subsequent flaring via lung as well as how manipulating cellular lipids through diet can be exploited to prevent environmental triggering of human autoimmune disease.

Key facts

NIH application ID
10167710
Project number
5R01ES027353-05
Recipient
MICHIGAN STATE UNIVERSITY
Principal Investigator
James J Pestka
Activity code
R01
Funding institute
NIH
Fiscal year
2021
Award amount
$416,246
Award type
5
Project period
2017-06-01 → 2022-12-14