OGT as a dosage sensor

NIH RePORTER · NIH · R01 · $415,928 · view on reporter.nih.gov ↗

Abstract

Project Summary Epigenetic regulators often lie downstream of signaling pathways that culminate in post-translational modifications, which affect their activity and ensure appropriate transcriptional responses to developmental and environmental cues. Our preliminary studies indicate that an interaction between epigenetic regulator, TET3, and an X-linked, post-translational modification enzyme, OGT, may be central in detecting X chromosome dosage and poising cells for X-inactivation. We propose to investigate the role of the TET3-OGT interaction in regulation of X chromosome inactivation, to obtain molecular insight into how cells count X chromosomes.

Key facts

NIH application ID
10200841
Project number
5R01GM128431-03
Recipient
UNIVERSITY OF CALIFORNIA, SAN FRANCISCO
Principal Investigator
ALMA L BURLINGAME
Activity code
R01
Funding institute
NIH
Fiscal year
2021
Award amount
$415,928
Award type
5
Project period
2019-09-15 → 2023-06-30