Inhibitory synaptic plasticity during learning

NIH RePORTER · NIH · R56 · $526,949 · view on reporter.nih.gov ↗

Abstract

Abstract What are the neural circuits by which the brain differentiates between incidental and meaningful environmental inputs to enable long-lasting changes in sensation and behavior? Experimental evidence indicates that this distinction may be made at the earliest stages of cortical processing, in primary sensory cortex. Here we will use high-throughput, automated behavioral training in freely-moving mice to determine how the detailed neural circuitry of the cerebral cortex is distinctly changed during acquisition of a tactile reward-based association. Our preliminary data indicate that long-lasting modifications in parvalbumin (PV)-mediated synaptic inhibition is selectively driven by sensory association training but not passive sensory exposure, providing a foothold to investigate the cellular circuitry that distinguishes between different types of experience-dependent plasticity. Using in vivo Ca imaging, targeted electrophysiological recordings and anatomical analyses, we will determine the mechanisms by which specific neural subtypes facilitate learning-related reorganization of the cortical column.

Key facts

NIH application ID
10270121
Project number
1R56NS119599-01
Recipient
CARNEGIE-MELLON UNIVERSITY
Principal Investigator
ALISON L BARTH
Activity code
R56
Funding institute
NIH
Fiscal year
2020
Award amount
$526,949
Award type
1
Project period
2020-09-30 → 2022-08-31