TSSK-dependent signaling pathway in spermatogenesis

NIH RePORTER · NIH · R03 · $161,500 · view on reporter.nih.gov ↗

Abstract

ABSTRACT Testis-specific serine kinases (TSSKs) belong to AMPK-related kinase family. Loss-of-function mutants of some TSSK members lead to spermatogenesis defects in male mice, and several SNPs of TSSKs are associated with male infertility in humans. However, the TSSK signaling mechanism during spermatogenesis is not understood. This proposal employs Drosophila as a model system to investigate a novel kinase-substrate pair: TSSKs-class IIa HDACs. Aim 1 will determine whether class IIa HDACs are substrates of TSSKs using both biochemical and genetic approaches. Aim 2 will explore the functional relevance of TSSKs-class IIa HDACs during spermatogenesis in Drosophila. Collectively, this proposal will establish class IIa HDACs as the key downstream component of TSSK signaling.

Key facts

NIH application ID
10450404
Project number
1R03TR003626-01A1
Recipient
UNIVERSITY OF CALIFORNIA, SAN FRANCISCO
Principal Investigator
Biao Wang
Activity code
R03
Funding institute
NIH
Fiscal year
2022
Award amount
$161,500
Award type
1
Project period
2022-06-01 → 2023-11-30