Understanding the role of the frontal cortex during cognitive flexibility in Fmr1 knock outs

NIH RePORTER · NIH · R03 · $71,589 · view on reporter.nih.gov ↗

Abstract

PROJECT SUMMARY Autism spectrum disorder (ASD) is a neurodevelopmental disorder where individuals exhibit deficits in cognitive flexibility. Purpose: This proposal will record from the frontal cortex of Fmr1 KOs (model of Fragile X syndrome) during cognitive flexibility tasks and assess how neural hyperexcitability within the KOs impacts reversal learning and shifts of attention. Methods: Fmr1 KOs and wildtype rats will be trained on categorical perception tasks with touchscreens in operant chambers. Single-unit neurophysiology and local field potentials will be collected from the frontal cortex (including the anterior cingulate) during the tasks. It is important to understand how altered neural function within the KOs impacts cognitive function and learning. This project will compare neural activity from putative pyramidal and interneurons to examine differential functions during cognition. Examining these differences in neural function between KOs and WT will enhance our understanding of how cortical excitability impacts cognition. The local field potentials can provide templates of neural signatures that in future studies can be compared to electroencephalography data from humans with ASD or Fragile X.

Key facts

NIH application ID
10795088
Project number
5R03MH133173-02
Recipient
KANSAS STATE UNIVERSITY
Principal Investigator
Bethany Plakke Anderson
Activity code
R03
Funding institute
NIH
Fiscal year
2024
Award amount
$71,589
Award type
5
Project period
2023-03-01 → 2026-02-28