PROMOTING T CELL RECOVERY AFTER RADIATION INJURY WITH LONG-ACTING INTERLEUKIN 7

NIH RePORTER · NIH · N01 · $1,999,087 · view on reporter.nih.gov ↗

Abstract

This proposal is designed to assess a long-acting interleukin 7 product, NT-I7, on phenotypic and functional T cell recovery after total body irradiation at radiation doses simulating exposures that would be expected during a radiation mass casualty incident. Clinical-grade NT-I7 is produced by NeoImmuneTech, Inc. (Rockville, Maryland), and is a recombinant human IL-7 immunoglobulin fusion protein that is more potent, stable, and longer acting than endogenous human IL-7. IL-7 is a cytokine involved in several areas of the adaptive immune system including thymopoiesis and NT-I7 acts as an exogenous form of IL-7 that exhibits beneficial effects on T cell recovery and increased antigen (Ag)-specific T cell response to viral infection. In preclinical studies NT-I7 has demonstrated beneficial preliminary results for T cell reconstitution after total body irradiation in mouse models. This is important because T lymphocytes are the most radiation sensitive blood cell type, a critical component of the immune response to infection – a significant risk following radiation injury – and there are no FDA approved drugs to support T cell recovery after radiation exposure.

Key facts

NIH application ID
11165542
Project number
75N93024C00005-0-9999-1
Recipient
DUKE UNIVERSITY
Principal Investigator
BENNY CHEN
Activity code
N01
Funding institute
NIH
Fiscal year
2024
Award amount
$1,999,087
Award type
Project period
2024-03-01 → 2025-02-28