Control of C. elegans lineage by heterochronic genes

NIH RePORTER · NIH · R01 · $648,090 · view on reporter.nih.gov ↗

Abstract

Project Summary/Abstract: We have identified a number of genes via genetic analysis and RNA interference gene inactivations that act as protein coding cofactors for the function of miRNAs and siRNAs in C. elegans. Some of these proteins were identified in genetic screens for decrease in miRNA function, some in genetic screens for decrease in siRNA function, and some in genetic screens for increase in siRNA function. We have also identified the target small RNAs that mediate these functions by deep RNA sequencing of selected mutant strains. We propose to dissect in detail how these proteins orchestrate the production, trafficking, and function of small RNAs in both mRNA degradation, mRNA translational control, and control of gene expression. We also propose to discern how the miRNA and siRNA and other small RNA pathways may compete with each other for common cofactors, thus leading to an increase in function in one pathway, when the other pathway is debilitated.

Key facts

NIH application ID
9815968
Project number
5R01GM044619-28
Recipient
MASSACHUSETTS GENERAL HOSPITAL
Principal Investigator
GARY B RUVKUN
Activity code
R01
Funding institute
NIH
Fiscal year
2020
Award amount
$648,090
Award type
5
Project period
1991-05-01 → 2021-04-30