Antibody Cooperation mediated by Fc-gamma Receptor (FcyR)-bearing cells

NIH RePORTER · NIH · P01 · $726,844 · view on reporter.nih.gov ↗

Abstract

Project 2. Antibody Cooperation for Fc-Fc-gamma Receptor (FcγR)-mediated functions. Although intrinsic differences in Fc-FcR interactions exist between humans and RM, we hypothesize that similarities between the two species for engagement of the FcγR-bearing cells can be defined to 1) select RM with FcγR genotypes/phenotypes mediating antiviral functions that match those observed in humans; and 2) identify combinations of antibodies of multiple specificities, through highly selective Fc- FcγR antibody interactions, that can act in concert to recruit Fc-gamma R-bearing cells similar to humans The overall Program hypothesis is that the RM model can be substantially improved for testing antibody-based interventions and vaccines through elucidation of key variables that impact species-specific FcgR-dependent effector functions (i.e. antibody epitope specificity, immune complex formation, isotype/subclass, glycosylation, and FcR genotype/phenotype. The specific aims for Project 2 are as follows: AIM 1. Define similarities and differences in FcγR mediated Ab effector function between RM and humans. AIM 2. Determine the combination of antibodies that together can mediate superior antiviral function

Key facts

NIH application ID
9925744
Project number
5P01AI120756-05
Recipient
DUKE UNIVERSITY
Principal Investigator
Guido Ferrari
Activity code
P01
Funding institute
NIH
Fiscal year
2020
Award amount
$726,844
Award type
5
Project period
— → 2024-04-30